Ulimi wangu jamani nisaidieni!

Ulimi wangu jamani nisaidieni!

Atakuwa aliutumbukiza sijui wapi.

Umenichekesha eti konie njoo, waatoto wa kileo wana majanga sana.

deep kissing???
hebu kwanza Kongosho aje!

hivi unakumbuka kisa cha yule mdada wa salenda aliyempa omba omba hela mkono ukaota manyoya kama wa paka??
 
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Atakuwa aliutumbukiza sijui wapi.

Umenichekesha eti konie njoo, waatoto wa kileo wana majanga sana.

mie nilishindwa kukoment manake deep kissing sijui ndo ikoje hivyo hadi ulimi uoteshwe nywele.
 
ukipiga mswaki huo uoto mweupe huwa unatoka?


Hairy leukoplakia (also known as oral
hairy leukoplakia,[1] :385 OHL, or HIV-
associated hairy leukoplakia),[2] is a white
patch on the side of the tongue with a
corrugated or hairy appearance. It is
caused by Epstein-Barr virus (EBV) and
occurs usually in persons who are
immunocompromised , especially those with
human immunodeficiency virus infection/
acquired immunodeficiency syndrome (HIV/
AIDS). This white lesion cannot be scraped
off, much like idiopathic leukoplakia. The
lesion itself is benign and does not require
any treatment, although its appearance may
have diagnostic and prognostic implications
for the underlying condition.

Signs and symptoms
There are no symptoms associated with the
lesion itself, [5] although many and varied
symptoms and signs may be associated with
the underlying cause of
immunosuppression. The lesion is a white
patch, which almost exclusively occurs on
the lateral surfaces of the tongue, although
rarely it may occur on the buccal mucosa ,
soft palate, pharynx or esophagus .[6] The
lesion may grow to involve the dorsal
surface of the tongue. The texture is
vertically corrugated ("hairy") or thickly
furrowed and shaggy in appearance.

Diagnosis
The white lesion cannot be wiped away, [8]
unlike some other common oral white
lesions, e.g. pseudomembranous
candidiasis, and this may aid in the
diagnosis. Diagnosis of OHL is mainly
clinical, but can be supported by proof of
EBV in the lesion (achieved by in situ
hybridization , polymerase chain reaction ,
immunohistochemistry, Southern blotting,
or electron microscopy ) and HIV
serotesting. [8] When clinical appearance
alone is used to diagnose OHL, there is a
false positive rate of 17% compared to
more objective methods. [4] The appearance
of OHL in a person who is known to be
infected with HIV does not usually require
further diagnostic tests as the association is
well known. OHL in persons with no known
cause of immunocompromise usually
triggers investigations to look for an
underlying cause. If tissue biopsy is carried
out, the histopathologic appearance is of
hyperlastic and parakeratinized epithelium,
with "balloon cells" (lightly staining cells) in
the upper stratum spinosum and "nuclear
beading" in the superficial layers (scattered
cells with peripheral margination of
chromatin and clear nuclei, created by
displacement of chromatin to the peripheral
nuclus by EBV replication). Candida usually
is seen growing in the parakeratin layer, but
there are no normal inflammatory reactions
to this in the tissues. [6] There is no
dysplasia (OHL is not a premalignant
lesion). [6]
 
No nkipiga mswak huwa hautoki but hiv nlshapima sina mkuu
 
pole sana, inaitwa oral thrust.. inapoteza raha sana na sometimes utakosa hamu ya kula. kama hadi kwenye lips basi iyo inakubidi ufike hospital bila kusahau kufanya usafi wa kinywa chako kila baada ya kula.. Ondoa hofu maana hofu hasa ya kuambukizwa maradhi huongeza tatizo la utando sehemu ya juu ya ulimi.. all the best
 
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