Nakaribisha maswali kuhusu Tiba / Dawa

Nakaribisha maswali kuhusu Tiba / Dawa

Mimi nataka kujua DOMS inasababishwa na nini na nini tiba yake.
 
Mimi nataka kujua DOMS inasababishwa na nini na nini tiba yake.
Delayed onset muscle soreness has up to six hypothesized theories have been proposed for the mechanism of DOMS, namely: lactic acid, muscle spasm, connective tissue damage, muscle damage, inflammation and the enzyme efflux theories.

However, an integration of two or more theories is likely to explain muscle soreness. DOMS can affect athletic performance by causing a reduction in joint range of motion, shock attenuation and peak torque. Alterations in muscle sequencing and recruitment patterns may also occur, causing unaccustomed stress to be placed on muscle ligaments and tendons.

These compensatory mechanisms may increase the risk of further injury if a premature return to sport is attempted.A number of treatment strategies have been introduced to help alleviate the severity of DOMS and to restore the maximal function of the muscles as rapidly as possible.

Nonsteroidal anti-inflammatory drugs have demonstrated dosage-dependent effects that may also be influenced by the time of administration. Similarly, massage has shown varying results that may be attributed to the time of massage application and the type of massage technique used.

Cryotherapy, stretching, homeopathy, ultrasound and electrical current modalities have demonstrated no effect on the alleviation of muscle soreness or other DOMS symptoms.

Exercise is the most effective means of alleviating pain during DOMS, however the analgesic effect is also temporary.
 
Delayed onset muscle soreness has up to six hypothesized theories have been proposed for the mechanism of DOMS, namely: lactic acid, muscle spasm, connective tissue damage, muscle damage, inflammation and the enzyme efflux theories. However, an integration of two or more theories is likely to explain muscle soreness. DOMS can affect athletic performance by causing a reduction in joint range of motion, shock attenuation and peak torque. Alterations in muscle sequencing and recruitment patterns may also occur, causing unaccustomed stress to be placed on muscle ligaments and tendons. These compensatory mechanisms may increase the risk of further injury if a premature return to sport is attempted.A number of treatment strategies have been introduced to help alleviate the severity of DOMS and to restore the maximal function of the muscles as rapidly as possible. Nonsteroidal anti-inflammatory drugs have demonstrated dosage-dependent effects that may also be influenced by the time of administration. Similarly, massage has shown varying results that may be attributed to the time of massage application and the type of massage technique used. Cryotherapy, stretching, homeopathy, ultrasound and electrical current modalities have demonstrated no effect on the alleviation of muscle soreness or other DOMS symptoms. Exercise is the most effective means of alleviating pain during DOMS, however the analgesic effect is also temporary.

Hahahaaa thank you for answering something I know very well.

But quick question. Did you write that or you got it from somewhere else?
 
Oh my bad.

I didn't see the link at first.

Thank you, kindly.
Its not easy to see cause its a special link for articles.Use PUBMED to get access to proper links/articles.
Im sorry i dont have alot of knowldge about the disease and there just alot of hypothesis around it so there is not much i can declare.As long as it a muscle weakness due to athelics i can only rely on muscle lactic acid and in comprehensive to DOMs it might be worse than that since the lactic acid varies more than a week and normal lactic acid should vary for two days.So there a lot of speculations around the disorder.I hope the information was useful.
 
So hiyo c.polymer in fanya kazi factor gani ktk mgando wa damu?

Blood coagulation factor zipo 13 (I-XIII) ambazo zinapitia blood clotting cascade.Factor VII inasaidia formation of fibrin which stops bleeding.
 
naomba unisaidie laboratory diagnosis & management ya VIBRIO PARAHAEMOLYTICUS
 
Nimesha jaribu maji ya aina zote, lakini tatizo liko pale pale!
 
naomba unisaidie laboratory diagnosis & management ya VIBRIO PARAHAEMOLYTICUS
  • Consumption is acquired from contaminated fish and sea food.
  • Clinical features are the same as Vibrio cholera 2-3 days incubation period,duration up to 7 days.
  • Symptoms are watery diarrhea,vomitting,abdominal cramps.
  • Laboratory identification:Grow in alkaline conditions(can be selected from other gut flora in alkaline peptone water).Oxidase positive.Grow on thiosulfate citrate bile salts sucrose(TCBS) medium to form green colonies.The Kanagawa test will complete the identification.
  • V.parahaemolyticus produces a heat-stable cytotoxin (which hemolyzes human red blood cells).Specific treatment not indicated for V.parahaemolyticus diarrhea but combination of tetracycline plus fluoroquinolone can be used.
  • Management is by adequate cooking of sea food.
 
maumivu hakuna ila kw mbali cna nasikia vitu vinachoma mzigo unapoanza kutoka dr
 
Dr ningependa kupewa elimu sahii juu ya side effect ya dawa za kurefusha maisha, maana nina case mbili ambazo zinanipa maswali mengi, kuna watu wawili tofauti katika vipindi tofauti wote baada ya kutumia dose iliwapelekea kupoteza kumbukumbu, kuumwa sana kichwa, kupofuka macho na mwisho kabisa kifo; je dozi hizo zinaweza kupelekea yote hayo endapo zisipotumiwa sahii?
 
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